基于PLC的烧结炉温度控制

基于PLC的温度控制系统以三菱FX2N型PLC为控制核心,由扩展模块FX2N-4AD-TC,FX2N-2DA,热电偶组成。根据已经建立的模糊语言变量赋值表和模糊控制规则,进行模糊推理和模糊判决,建立离线的模糊控制查询表,并给出电阻炉温度模糊控制的相关程序。采用Fuzzy-PID复合控制,系统同时获得良好的动态性能和稳态性能。     

已下挠连续梁桥加固方案研究

针对目前一些大跨径预应力混凝土连续梁桥跨中长期挠度过大的问题,以汾江大桥为工程背景,分析导致该桥主梁挠度过大的主要原因。通过实测资料进行计算分析,说明体外预应力加固未能阻止主梁进一步下挠的原因。提出一种以钢箱梁替代跨中梁段的加固方案,通过计算分析其加固效果并与体外预应力加固进行对比,简单分析其可行性并展望应用前景。     

基于粗糙集-模糊识别技术的交通流状态识别算法研究

构建了基于粗糙集约简的交通流状态模糊识别算法,利用粗糙集属性约简技术在多个交通流参数中获得反映交通流变化的表征变量及其重要度值,在此基础上采用模糊识别方法构建交通流状态识别算法,算法针对三种交通流状态的识别,包括正常状态、常发性拥堵状态、偶发性拥堵状态.利用实际城市快速路的数据,以识别率和误报率为衡量指标,将文中的算法与其他算法进行了对比研究,结果表明在同样的误报率水平下可以得到更高的识别率.     

实时交通信息提供对驾驶员路径选择行为影响量化分析

研究的目的是搞清交通信息提供对驾驶员路径选择行为的影响机理和程度,为智能交通系统(ITS)中交通诱导方案的制定提供参考与依据.研究采用SP(Stated Preference Survey)调查方法针对驾驶员对交通信息响应状况进行调查,同时对目前交通信息提供现状进行了调查.对调查数据进行定性和定量分析的基础上,建立了信息提供下驾驶员路径选择行为的Logistic模型,可用于判别分析.研究发现各变量在不同程度上影响驾驶员路径改变倾向,其中驾驶员对交通信息广播的准确性评价以及能够容忍延误时间的程度等因素对驾驶员路径选择行为影响较大.     

舰船水压场信号实时检测方法研究

为了有效地从风浪背景中检测舰船水压场信号,在对大量实测海浪水压场数据和舰船水压场数据分析的基础上,对海浪水压场信号的特性进行了研究,发现海浪水压场信号可用AR模型对其进行描述．从而对海浪水压场信号建立AR模型,计算AR模型的反射系数并提取其欧氏距离作为特征,采用滑动检测法对信号进行实时检测．通过大量实测数据检验其有效性,结果表明,该算法简单,能较好的检测目标信号．     

考虑引桥工作状态下的短跨板桥的车致动力反应

通过把车辆和桥梁看作一个耦合系统来建立模型去求解桥梁的车致动力反应。文中研究了由AASHTO HS20卡车引起的不同跨径的短板桥的动力反应。利用参数分析来研究不同车速及不同路面状况所产生的效应。研究发现,引起桥梁动力反应峰值的临界车速遵循描述列车致桥梁共振的规则。分析中考虑了引桥状态的影响包括引桥桥头断层及引桥板形变。虽然引桥变形对桥梁的动力反应影响微小,但是在研究中却发现引桥桥头断层能在短跨板桥中引起显著的动力反应。该文对评价既有短跨径桥梁的动力性能有很大的意义,因此值得中国从业者借鉴。     

花石板1号大桥跨越特大型溶洞的处理方案

山区高速公路桥梁、隧道施工遇到特大型溶洞时,必须进行加固处理。以沪溶国道某桥为例,提出顶板锚喷加立柱支撑的处理方案并进行稳定性评价。实践证明,处理方案合理、经济、安全。     

达克罗涂层对STR车轮螺栓防腐性能的影响

通过对STR车轮螺栓的磷化、镀锌和达克罗处理,多角度分析比较了三种处理方式的优劣,结果表明:经达克罗及其后处理后的螺栓和螺母具有良好的润滑性、硬度和耐腐蚀性,试验前螺栓的拆卸次数越多,盐雾试验后其锈蚀程度越严重,其中达克罗螺栓的临界拆卸次数为10次;另外,本文展望了达克罗绿色环保特性在国内汽车业的发展前景。     

Induction of animal model of Graves' disease in BALB/c mice

Objective To construct an animal model of Graves' disease (GD) by immunizing BALB/c mice with hM12 cells co-expressing major histocompatibility complex (MHC) class Ⅱ molecules and human thyrotropin receptor (TSHR) molecules. Methods BALB/c mice in experimental group (H-2d) were immunized with hM12 cells Intraper-itoncally every 2 weeks for six times, while mice in control group were immunized with M12 cells. Five weeks later, the thyroids were histologically examined, and serum samples were tested for thyroid-stimulating antibodies (TSAb) and thyroid hormone levels. Results One BALB/c mouse in experimental group developed Graves'-like disease. Total T4 and T3 levels in this mouse were above the upper limit of normal, TSAb activity was displayed in its serum. The thyroid histologically showed the features of thyroid hyperactivity including thyrocyte hypercellularity and colloid absorption.None of control mice developed Graves'-like disease. Conclusion An animal model with some characteristics of human Graves' disease was successfully induced and the model will facilitate studies aimed directly at understanding the patho-genesis of autoimmunity in Graves' disease.     

Glutamate enhances the expression of vascular endothelial growth factor in cultured SD rat astrocytes

Objective To study the effect of glutamate on the expression of vascular endothelial growth factor (VEGF) mRNA and protein in cultured rat astrocytes. Methods Cultured rat astrocytes were randomly divided into 6 groups: control group (C), glutamate group (G), QA group (Q), DCG-IV group (D), L-AP4 group (L) and glutanmte-FMCPG gronp (G+M). Cells were cultured under nomoxic condition (95% air, 5% CO2). RT-PCR and ELISA methods were used to detect the expression of VEGF mRNA and protein in cultured astrocytes, respectively. G+ M group was preincubated with lmM MCPG for 30 min prior to the stimulation with glutamate. There were 7 time points at 0,4,8,12,16,24 and 48 h in each group except G+M group. Results The expression of VEGF mRNA and protein did not differ significantly among D group, L group and C group. Different from that in C group, the expression of VEGF mRNA and protein could be enhanced both in a dose-dependent and time-dependent manner in G group and Q group. Meanwhile, the enhanced expression of VEGF mRNA and protein in G group was completely suppressed by MCPG after 24 h. Conclusion Glutamate can increase the expression of VEGF mRNA and protein in cultured astrocytes, which may be due to the activation of group I metabotropic glutamate receptors in astrocytes.