Beyond their functional purpose, cars are often considered a status symbol. There may exist a certain level of pride associated with owning and using cars, particularly in regions where motorization is rapidly growing. However, there is little empirical evidence in terms of how car pride is related to different behavioral aspects, such as car ownership and use, especially in the context of developing countries. This paper presents an exploration of car pride and its association with car-related behavior. In this work, car pride is defined as the self-conscious emotion derived from the appraisal of owning and using cars as a positive self-representation. It pertains to both the symbolic and affective functions of the car. Using survey data (n?=?1389) from Shanghai, China, we empirically measure car pride as a latent variable based on five Likert-scale statements and test the association of car pride with car use, vehicle preferences, and car ownership. Based on two structural equation models, we show that: (1) car pride is positively correlated with car use; (2) car pride correlates significantly with owning newer, more expensive, and luxury cars, and Shanghai’s more expensive local car licenses; (3) car owners in general have higher car pride than non-owners; and (4) car pride is largely independent of one’s socio-economic characteristics. Although the analysis focuses on Shanghai, the findings of the positive correlation between car pride and behavior are consistent with prior studies in developed countries. These findings highlight the importance of car pride regarding multiple behavioral aspects of car ownership and use and its potential impact on mobility management.
Objective To explore the influence of the acute cauda equina compression on the iumbosacral spinal cord; To clarify the pathologic change of the motor neuron after acute cauda equina compression. Methods 27 canis familiaris were randomly divided into 9 groups (3 in each) : one for normal group, one for control group, and seven for compression groups. The control group and compressed groups was given operation and the sac made of silica gel was placed under the lamina of L5-6. Water was injected into the sac until their posterior legs paralysis in compressed groups, the animals had been compressed for 4, 8, 12, 24, 48, 72, 168 hours. The control group were not injected water. Cells apoptosis was investigated with the technology of TdT-mediated biotin dUTP nick end-labeling (TUNEL) staining. The Bcl-2 Bax and Caspase-3 protein was investigated by immunohistochemical method. Results TUNEL staining cells in anterior horn presented after compressed 8-12 hours, and at 72 hours the number of positive cells got to maximum, it decreased subsequently after 168 hours. The protein of Bax, Bcl-2 expressed a little in normal motor neuron. The caspase-3 protein didn't express in normal ceil. They all reached the peak at 72 hours after compression. Conclusion The apoptosis of motor neuron occurred earlier after eauda equina acute compression. Bax protein restrained Bcl-2 protein then active caspase-3 and conduced apoptosis of motor neuron. 相似文献