基于多源数据的公交车能耗碳排放测算模型

公交车能耗碳排放强度与车辆、线路和驾驶员有显著相关关系，为精准刻画其能耗碳排放强度特征，整合OBD监测数据、加油(气)数据、运营排班数据等多源数据资源. OBD监测数据和加油(气)数据呈显著的线性关系，证明修正后的OBD监测数据可满足分析要求. 搭建“速度-能耗碳排放强度曲线”测算模型，幂函数关系的拟合优度R2 =0.972 6 为最高. 实证研究发现，平均速度在10~60 km/h 变化时，液化天然气(LNG)车比柴油车能耗碳排放强度高 3.3%~33.7%，双层车比铰接车高2.4%~13.3%；LNG铰接车在不同线路、相同速度下的强度相差9.6%；不同驾驶员在相同线路的能耗碳排放强度可相差24.2%. 模型为各城市基于多源数据开展公交能耗碳排放目标设定提供数据支撑.     

A cost-competitiveness analysis of charging infrastructure for electric bus operations

This study investigates the cost competitiveness of different types of charging infrastructure, including charging stations, charging lanes (via charging-while-driving technologies) and battery swapping stations, in support of an electric public transit system. To this end, we first establish mathematical models to investigate the optimal deployment of various charging facilities along the transit line and determine the optimal size of the electric bus fleet, as well as their batteries, to minimize total infrastructure and fleet costs while guaranteeing service frequency and satisfying the charging needs of the transit system. We then conduct an empirical analysis utilizing available real-world data. The results suggest that: (1) the service frequency, circulation length, and operating speed of a transit system may have a great impact on the cost competitiveness of different charging infrastructure; (2) charging lanes enabled by currently available inductive wireless charging technology are cost competitive for most of the existing bus rapid transit corridors; (3) swapping stations can yield a lower total cost than charging lanes and charging stations for transit systems with high operating speed and low service frequency; (4) charging stations are cost competitive only for transit systems with very low service frequency and short circulation; and (5) the key to making charging lanes more competitive for transit systems with low service frequency and high operating speed is to reduce their unit-length construction cost or enhance their charging power.     

Dynamic demand estimation and prediction for traffic urban networks adopting new data sources

Nowadays, new mobility information can be derived from advanced traffic surveillance systems that collect updated traffic measurements, both in fixed locations and over specific corridors or paths. Such recent technological developments point to challenging and promising opportunities that academics and practitioners have only partially explored so far.The paper looks at some of these opportunities within the Dynamic Demand Estimation problem (DDEP). At first, data heterogeneity, accounting for different sets of data providing a wide spatial coverage, has been investigated for the benefit of off-line demand estimation. In an attempt to mimic the current urban networks monitoring, examples of complex real case applications are being reported where route travel times and route choice probabilities from probe vehicles are exploited together with common link traffic measurements.Subsequently, on-line detection of non-recurrent conditions is being recorded, adopting a sequential approach based on an extension of the Kalman Filter theory called Local Ensemble Transformed Kalman Filter (LETKF).Both the off-line and the on-line investigations adopt a simulation approach capable of capturing the highly nonlinear dependence between the travel demand and the traffic measurements through the use of dynamic traffic assignment models. Consequently, the possibility of using collected traffic information is enhanced, thus overcoming most of the limitations of current DDEP approaches found in the literature.     

城市交通公平性分析及对策

城市道路的交通公平性有利于建设和谐、公平的城市,在交通公平的定义以及它的三层含义的基础上,陈述了交通公平在时间、空间和内容上的内涵。分析城市道路交通公平的影响因素及其判断标准,阐述了快速公交符合城市道路交通公平的判断标准是它有利于大多数人的出行,满足弱势群体的出行要求,并应具有低成本、高质量的优点。最后,以北京市BRT的实践表明快速公交是提高城市交通公平性的有效措施。     

关于公交停靠站影响区范围的研究

随着城市公共交通系统的发展,公交停靠站对道路交通流的影响已逐渐引起人们的注意。分析公交停靠站的影响首先应该对停靠站的影响区范围进行界定。在分析前人研究成果的基础上,基于交通波理论,按考虑超车与否,分析公交停靠站的影响区范围长度,得到不同交通流密度条件下公交停靠站影响区长度的计算公式。     

快速老化小鼠的学习记忆缺陷及其生化机制

目的用快速老化小鼠(SAMP8)模拟老年痴呆,观察其学习记忆能力,并探讨其学习记忆能力下降的可能机制。方法采用10月龄SAMP8,并用同龄抗快速老化小鼠(SAMR1)作为正常对照。用水迷路检测近记忆以及空间学习记忆能力。用生物化学方法检测大脑皮层线粒体膜电位和ATP酶活力,观察线粒体功能;检测皮层和海马胆碱乙酰基转移酶(ChAT)和乙酰胆碱酯酶(AChE)的活力,观察中枢胆碱能神经功能;检测血清超氧化物歧化酶(SOD)活力和丙二醛(MDA)含量,观察其全身性氧化应激状况。结果与SAMR1相比,SAMP8出现明显的衰老体征,其近记忆和空间学习记忆能力明显减弱,具有衰老和痴呆特征;皮层线粒体膜电位和ATP酶活力显著降低,海马ChAT活力显著下降,AChE活力显著升高,血清SOD活力稍有增高。结论SAMP8可以很好地模拟老年痴呆,其机制包括大脑皮层线粒体功能降低、海马胆碱能神经功能下降以及氧化应激等。     

城市公交GPS轨迹路线图制作研究

以哈尔滨市104路公交为主要研究对象,详细介绍城市公交的GPS轨迹路线图制作时所需的软硬件,以及数据的采集方法,叙述数据的整合与分类过程。通过将主要公交站点周围兴趣点信息增添到轨迹点附近,构成包含有站点轨迹信息、站点影像信息的沿途轨迹信息。经过整合与分类,通过HOLUX ezTour软件对数据进行处理,阐述轨迹查询、地标的新增与删改和轨迹的添加与变换等功能。并使用Google Earth三维图像对轨迹路线全面整合,最终制作成城市公交GPS轨迹路线图。     

基于ASP.NET的B/S架构的软件在Windows7系统下的部署

本文重点研究了对于B/S架构的软件在Windows7系统下部署时应该采用的一系列方法及在部署时遇到的一系列问题的解决方案。文中分析了利用ASP.NET开发的B/S架构的软件的优点并在此基础上介绍了软件部署的流程。本文在分析过程中结合具体实例提出了部署中一些常见问题的解决方案。     

姜黄素促进RAW264.7源性M1巨噬细胞向替代激活M2表型极化

目的观察姜黄素对LPS和IFNγ诱导的RAW264.7巨噬细胞(M1)极化的影响及机制。方法不同浓度姜黄素(6.25、12.5、25μmol/L)干预LPS和IFNγ诱导的RAW264.7巨噬细胞(M1)12h,同时再加入20μmol/L GW9662与25μmol/L姜黄素共同干预LPS和IFNγ诱导的RAW264.7巨噬细胞(M1)12h,采用Real-time PCR、ELISA及Western blot方法检测IL-1β、IL-6和M2表型标志分子KLF4、FIZZ1、MGL1、PPARγ的表达,及阻断PPARγ后KLF4和FIZZ1的表达。结果不同浓度姜黄素均能上调LPS和IFNγ诱导的RAW264.7巨噬细胞(M1)的M2标志分子的表达,并且抑制炎症因子IL-1β和IL-6的分泌;阻断PPARγ后,RAW264.7巨噬细胞源性M1表型巨噬细胞表达M2标志分子下调。结论姜黄素通过活化PPARγ促进LPS和IFNγ诱导的RAW264.7巨噬细胞(M1)向M2表型极化。     

吸烟和β3-肾上腺素能受体基因Trp64Arg、锰超氧化物歧化酶9Ala/Val基因多态性与非酒精性脂肪性肝病的相关性

目的探讨吸烟和β3-肾上腺素能受体(β3-AR))基因Trp64Arg、锰超氧化物歧化酶9Ala/Val(MnSOD9Ala/Val)基因多态性与非酒精性脂肪性肝病(NAFLD)发病之间的关系。方法采用病例-对照研究的方法,以720例NAFLD患者及720例健康对照者的外周血白细胞为样本,采用聚合酶链反应(PCR)技术分析β3-AR基因Trp64Arg和MnSOD9Ala/Val基因多态性。结果β3-AR基因Trp64Arg(A/A)基因型和MnSOD9Ala/Val(V/V)基因型频率分布分别为39.4%、71.7%(病例组)和21.1%、43.3%(对照组),差异有统计学意义(P<0.01;P<0.01)。Trp64Arg(A/A)基因型者患NAFLD的风险显著增加(OR=2.434,95%CI=1.816~4.075)。MnSOD9Ala/Val(V/V)基因型者患NAFLD的风险也显著增加(OR=3.308,95%CI=1.913~4.509)。基因突变的协同分析发现,Trp64Arg(A/A)/MnSOD9Ala/Val(V/V)基因型者在NAFLD组和对照组中的分布频率分别为32.8%和6.5%,差异有统计学意义(P<0.01)。Trp64Arg(A/A)/MnSOD9Ala/Val(V/V)基因型者患NAFLD的风险显著增加(OR=9.753,95%CI=4.292~12.426)。病例组的吸烟率显著高于对照组(OR=2.623,95%CI=1.425~4.957),Trp64Arg(A/A)/MnSOD9Ala/Val(V/V)基因型与吸烟有协同作用(OR=33.764,95%CI=18.907~61.582)。结论 Trp64Arg(A/A)/MnSOD9Ala/Val(V/V)基因型和吸烟是NAFLD的易患因素,三者的联合在NAFLD的发生中起着协同的作用。