全文获取类型
收费全文 | 1050篇 |
免费 | 62篇 |
专业分类
公路运输 | 293篇 |
综合类 | 660篇 |
水路运输 | 39篇 |
铁路运输 | 102篇 |
综合运输 | 18篇 |
出版年
2024年 | 1篇 |
2023年 | 4篇 |
2022年 | 8篇 |
2021年 | 11篇 |
2020年 | 34篇 |
2019年 | 9篇 |
2018年 | 17篇 |
2017年 | 8篇 |
2016年 | 5篇 |
2015年 | 21篇 |
2014年 | 95篇 |
2013年 | 77篇 |
2012年 | 95篇 |
2011年 | 80篇 |
2010年 | 82篇 |
2009年 | 60篇 |
2008年 | 87篇 |
2007年 | 82篇 |
2006年 | 77篇 |
2005年 | 43篇 |
2004年 | 17篇 |
2003年 | 26篇 |
2002年 | 24篇 |
2001年 | 17篇 |
2000年 | 16篇 |
1999年 | 14篇 |
1998年 | 10篇 |
1997年 | 11篇 |
1996年 | 6篇 |
1995年 | 11篇 |
1994年 | 14篇 |
1993年 | 7篇 |
1992年 | 9篇 |
1991年 | 11篇 |
1990年 | 4篇 |
1989年 | 8篇 |
1988年 | 3篇 |
1987年 | 4篇 |
1986年 | 2篇 |
1984年 | 1篇 |
1982年 | 1篇 |
排序方式: 共有1112条查询结果,搜索用时 281 毫秒
11.
水泥混凝土路面质量病害问题探讨 总被引:2,自引:0,他引:2
孙兴嫒 《筑路机械与施工机械化》2005,22(2):35-37
根据目前水泥混凝土路面建设比较普遍的情况,针对施工中出现比较多的质量病害进行了分析,分别从水泥混凝土路面的裂缝产生的过程,材料、配合比、水灰比、施工操作等对质量的影响,以及产生外观质量缺陷的原因进行了评述. 相似文献
12.
随着交通量的迅猛增长,桥梁单板受力情况越来越频繁,通过对单板受力现象产生原因进行分析,提出解决措施,以供同行参考和借鉴。 相似文献
13.
黄燕 《内蒙古公路与运输》2013,(4):77-78
结合博乐公路管理局管养的G30线K3981~K3982路段路基边坡防沙害处治养护项目的实施,对处治沙害实施流程进行了详细的阐述,有一定的参考价值。 相似文献
14.
大骨节病病因与发病机制的研究进展及其展望 总被引:13,自引:2,他引:11
郭雄 《西安交通大学学报(医学版)》2008,29(5)
大骨节病是一种地方性、畸形性骨关节病,至今原因未明,目前以我国西部地区最重,尚无有效治疗措施。本文依据国内外有关研究,总结分析了大骨节病三种病因假说的研究进展及其尚待解决的问题,结合作者及其研究团队近年来的研究进展,提出大骨节病除经典的软骨细胞坏死外还表现有软骨细胞过度凋亡和"去分化",大骨节病软骨细胞差异表达基因、血清蛋白表达谱及短串联重复序列多态性不同于病区内外正常人与骨关节病。确定大骨节病软骨细胞坏死发生的基因与蛋白标志物,以及何种环境因素如何与大骨节病易感基因相互作用而选择性的损伤深层软骨细胞及其干预措施,是未来大骨节病病因发病机制研究的重要目标。 相似文献
15.
崔卫东 《广东交通职业技术学院学报》2008,7(2):14-16
以105国道新丰段路面大修工程为依据,通过对旧路面的调查,分析了水泥砼路面损坏的原因,并采用了对应的处治措施。 相似文献
16.
Objective To study the changes in neuronal activity of the zone incerta (ZI) following the unilateral lesion of the nigrostriatal pathway. Methods Eiectrophysiological recordings of ZI neurons were made in normal rats and in two groups of rats at different time intervals after injection of 6-hydrodopamine (6-OHDA) into the pars compacta of snbstantia nigra by extracellular recording in vivo. Results The results showed a significant increase in the mean firing rate of ZI during the second and fourth weeks after 6-OHDA lesion [-n=32, (3.6±2. 2)Hz, P<0.001; n=35, (9.3±6.6)Hz, P<0. 001, respectively] compared to that of normal rats [n=39, (9.2±5.2)Hz]. However, no significant change was observed between two groups of 6-OHDA-lesioned rats. With regard to firing pattern, 7.7%(3/39) of ZI neurons discharged regularly, 82.1% (32/39) irregularly and 10. 3% (4/39) in bursts in normal rats. During the second week after 6-OHDA lesion, the regular, irregular and bursting firing neurons recorded in the ZI were 9.4% (3/32), 59. 4% (19/32) and 31.3% (10/32), and during fourth week, the regular, irregular and bursting firing neurons were 14.3% (5/35), 57.1% (20/35) and 28. 6% (10/35), respectively. The firing pattern of the neurons in the three different groups did not change significantly. Conclusion These results suggest that the firing rate of ZI neurons in 6-OHDA-lesioned rats is increased significantly, which may contribute to the pathophysiology of Parkinson's disease. 相似文献
17.
Objective To explore the risk association of ABCA1-V771M polymorphism with coronary heart disease (CHD) in Hart nationality in Northwest of China. Methods With case-control study, ABCA1-V771M polymorphism was detected in 204 unrelated Hart nationality people in Northwest of China, and all the subjects by coronary angiography were grouped into 106 cases and 98 controls. The genotypes and alleles frequency distribution of ABCA1-V771M polymorphisms were analyzed by PCR-RFLP analysis, and the clinical statistics of serum lipids were compared and its effects of ABCA1-V771M polymorphism on the plasma lipid levels and coronary atherosclerotic heart disease were analyzed. Results The genotypic frequencies of ABCA1-V771M polymorphism matched well under Hardy-Weinberg equilibrium (P>0.05), V and M allelic frequencies were 33.3% and 66.7%. In comparison with VV VM genotype carriers, MM genotypes carriers had much lower plasma levels of HDL-C (P<0. 001) and much higher plasma levels of TG (P<0. 05). M allelic frequency in CHD group was significantly higher than V allelic frequency (P<0. 05). M allele was related with more severity of atherosclerosis in the coronary artery than V allele (P<0.05). However, there was no obvious difference in the incidence of AMI among carriers with three genotypes of ABCA1-V771M polymorphism (P>0.05). Conclusion ABCA1-V771M polymorphism was not only associated with the plasma levels of HDL-C and TG, but also related to the susceptibility and severity of coronary atheroselerotic heart disease. Moreover, M771 allele appeared to be atherogenie among Han population in Northwest of China. 相似文献
18.
Objective To investigate changes in T lymphocyte subsets and NK cells in patients with simple Graves' disease (GD)and Graves' disease combined with type 2 diabetes mellitus (GD/T2DM). Methods Fifteen cases of GD/ T2DM were selected from our hospital from November 2001 to November 2004. Before and after therapy thyroid function, thyroglobulin antibody (TGA), thyroid microsomal antibody (TMA) and blood glucose level were measured, and T lymphocyte subsets (CD3, CD4, CD8, CD4/CD8) and NK cells (CD56) were measured by immunofluorescence double labeling monoclonal antibody and flow cytometry, respectively. At the same time, comparison was made with simple GD (15 cases), T2DM (15 cases) and healthy control (20 cases). Results Before therapy, CD4/CD8, CD4 and NK cells in GD/T2DM were less than normal, and there was no significant difference in comparison with simple GD (P<0.05). In T2DM group, only CD4/CD8 and CD4 were less than those of healthy controls (P<0.05). When thyroid function recovered after 1 to 3 months of methimazole treatment in both GD/T2DM and simple GD groups, various indexes recovered, which were more obvious in simple GD. Conclusion Immune hypofunction of GD may be the key to the immune abnormality of GD/T2DM, which is more significant than that of simple GD or T2DM. The recovery of thyroid function and immune abnormality is not consistent, and the recovery of GD is more significant than that of GD/T2DM. 相似文献
19.
20.
介绍桁式组合拱桥的典型病害,研究该类桥梁病害的成因,建立桁式组合拱桥的有限元模型,并进行结构的静力、动力分析。结合现场检测结果的结构病害,分析桁式组合拱桥典型病害产生的根源。根据该类桥梁的病害成因,提出几种桁式组合拱桥典型病害的处治方案。最后,通过实际的桁式组合拱桥检测加固工程,验证加固效果。通过对桁式组合拱桥典型病害成因分析和处治方法研究,为该类桥梁的加固建设提供借鉴。 相似文献